5,6-Methylenedioxy-2-aminoindane�(also known as�MDAI) is a lesser-known novel�entactogen�substance of the�aminoindane�class.mdai research,buy mdai,mdai for sale,mdai effects,mda
Notably, this compound primarily produces the non-stimulating effects of prototypical entactogens like�MDMA�such as�sedation,�muscle relaxation, and�tactile enhancement.
MDAI was developed in the 1990s by a team led by�David E. Nichols�at Purdue University.
It acts as a putatively non-neurotoxic and highly selective�serotonin�releasing agent�(SSRA) with neglible effects on�dopamine�and�norepinephrine.
This reportedly limits its potential at producing overtly invigorating, prosocial or�euphoric�effects.
MDAI has been marketed alongside�research chemical�entactogens�like�5-MAPB,�5-APB, and�6-APB�as a legal, grey-market alternative to�MDMA.
Very little data exists about the pharmacological properties, metabolism, and toxicity of MDAI, and it has a limited history of human use. It is highly advised to use�harm reduction practices�if using this substance.
MDAI, or 5,6-methylenedioxy-2-aminoindane, is a synthetic molecule of the�aminoindane�class with structural similarity to�amphetamines.mdai research,buy mdai,mdai for sale,mdai effects,mda
It features the R3�terminal carbon of the propane chain of amphetamine bound to the benzene ring.
This creates an indane group, a bicyclic moeity containing a benzene ring fused to a pentane ring. MDAI contains an amino group NH2�bound to R2�of the indane ring.
MDAI also contains two oxygen substitutions at R5�and R6 joined by a methylene bridge to form a methylenedioxy group. MDAI is structurally related to�2-AI, differing by a methylenedioxy ring.
MDAI has been shown to�inhibit the reuptake�of�serotonin�and has a selective affinity for the serotonin�receptor. Studies have shown that the brains of animals treated with MDAI have greater extracellular concentrations of�monoamine neural transmitters, most significantly�serotonin.
For comparison, MDAI is similar in potency with releasing serotonin to�MDA, but significantly less potent than�MDMA.
This is done by inhibiting the reuptake and reabsorption of the neurotransmitters after they have performed their function of transmitting a neural impulse, essentially allowing them to accumulate, be reused and cause�entactogenic�effects.
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